That's why that dumbass article about how locavores are preventing agricultural development was such nonsense. I'm not buying local food because it's going to save the world, though maybe it will. I'm buying it because it's better and it goes from the farmer's hands to mine or a butcher I trust. Farmers invite me to see their farms and I've even helped "process" chickens several times.
I guess that's our fundamental difference. I subscribe to the precautionary principle. News reports like this one are nothing new, and they are certainly evidence of potential risk of harm.
The developing fetus and neonate are particularly vulnerable to POPs exposure due to transplacental and lactational transfer of maternal burdens at critical periods of development. It has also been reported that residents of the Canadian Arctic, and who exist at the highest trophic level of the Arctic aquatic food chain, have PCB intake levels in excess of the acceptable daily intake, and that may place this population at special risk for reproductive and developmental effects. In another report, children in the northern Quebec region of Canada who have had significant exposure to PCBs, dioxins and furans through breast milk also had a higher incidence of middle ear infections than children who had been bottle fed. Most authors, however, conclude that the benefits of breast feeding outweighs the risks.
Studies of carcinogenesis associated with occupational exposure to 220.127.116.11-TCDD also seem to
indicate that extremely high-level exposures of human populations do elevate overall cancer
incidence. Laboratory studies provide convincing supporting evidence that selected
organochlorine chemicals (dioxins and furans) may have carcinogenic effects and act as strong
More recently, literature has been accumulating in which some researchers have suggested a
possible relationship between exposure to some POPs and human disease and reproductive
dysfunction. Researchers have suggested that the increasing incidence of reproductive
abnormalities in the human male may be related to increased estrogen (or estrogenic type)
compound exposure in vitro, and further suggest that a single maternal exposure during
pregnancy of minute amounts of TCDD may increase the frequency of cryptorchidism in male
offspring, with no apparent sign of intoxication in the mother. Associations have been made
between human exposure to certain chlorinated organic contaminants and cancers in human
populations. Preliminary evidence suggests a possible association between breast cancer and
elevated concentrations of DDE. While the role of phytoestrogens and alterations in lifestyle
cannot be dismissed as important risk factors in the dramatic increase in estrogen dependent
breast cancer incidence, correlative evidence suggesting a role for POPs continues to mount. This
latter theory has been supported in a report that noted that levels of DDE and PCBs were higher
for breast cancer case patients than for control subjects, noting that statistical significance was
achieved only for DDE. While a causal relationship between organochlorine exposure and
malignant breast disease remains far from proven, the possibility thatchronic low level exposure,
when coupled with the known bioaccumulative properties of POPs, may even contribute in some
small way to overall breast cancer risk has extraordinary implications for the reduction and
prevention of this very important disease.
They also reported that the rats on HFCS 24 hrs/day did not gain a statistically significant amount of weight when compared to the rats on sucrose or chow only. Additionally, no differences in blood-glucose levels were observed. Another study was conducted for 6-7 months, and fat pads were removed from the rats and weighed. Fat pads for rats on HFCS 12 hrs/day weighed significantly more than rats on chow only, but were not different from rats on sucrose. Fat pads for rats on HFCS 24 hrs/day did not have a statistically different weight than rats on chow only.
A 2008 study in humans analyzed the circulating levels of glucose, insulin, leptin, ghrelin, and triacylglycerol during a 24 hour period after consuming drinks containing HFCS or sucrose. The researchers concluded that the consumption of HFCS or sucrose did not yield differing metabolic effects.
The first study showed that male rats given water sweetened with high-fructose corn syrup in addition to a standard diet of rat chow gained much more weight than male rats that received water sweetened with table sugar, or sucrose, in conjunction with the standard diet. The concentration of sugar in the sucrose solution was the same as is found in some commercial soft drinks, while the high-fructose corn syrup solution was half as concentrated as most sodas.
The second experiment -- the first long-term study of the effects of high-fructose corn syrup consumption on obesity in lab animals -- monitored weight gain, body fat and triglyceride levels in rats with access to high-fructose corn syrup over a period of six months. Compared to animals eating only rat chow, rats on a diet rich in high-fructose corn syrup showed
I mean, this right here? This is a bloody hunk of cow muscle. This is, by any objective standard, entirely gross, and far more disgusting than ingesting things which, under other circumstances, might be proscribed you by a physician.
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